By Andrew Blann, Nessar Ahmed
This booklet is approved by means of the Institute of Biomedical technological know-how and written by means of its specialist writers, a lot of whom paintings at the Institute’s advisory panels.
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Additional resources for Blood Science: Principles and Pathology
High total cholesterol) as a risk factor for atherosclerosis is to fail to understand the value of measuring LDL and HDL. This is why we now prefer the word ‘dyslipidaemia’, as it does not focus on LDL or HDL. It is still a matter of debate as to whether or not isolated high concentrations of triacylglycerols are an independent risk factor of atherosclerosis in the general population. As we shall see, there are abnormalities in lipid metabolism when disease is associated with high triacylglycerols, but these are uncommon.
A second, allied, haemorrhagic condition is von Willebrand’s disease, caused by one of several mutations in the gene for von Willebrand factor. These mutated genes have varied penetration into the disease. In some, penetrance is high and the disease is severe (complete absence of von Willebrand factor). However, in others, the mutation causes a partial reduction in concentrations of the protein, and so minor bleeding, or the disease may even be largely asymptomatic, in which case penetrance is low.
We have already mentioned platelets are part of the full blood count. The clot (or thrombus) is generated by platelets and fibrin, which together form a net that traps red cells; but clots can occur without red cells. The laboratory offers tests on the ability of the blood to generate these clots. The inappropriate formation of a clot can lead to a disabling deep vein thrombosis or, more seriously, a stroke or even, in the lung, death. Conversely, inability to form a clot can also lead to serious disease due to excessive bleeding (haemorrhage).
Blood Science: Principles and Pathology by Andrew Blann, Nessar Ahmed